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The reason you went through all this work is to create a named range that automatically expands. Check to make sure your dynamic range expands downward and to the right. Type 2007 to the right of 2006 in the headers. Type the name of your county in the last cell of column B, below the California counties. Press F5, type in dbMdnHseInc, and see whether the range expands to include the new column and row. If it does, you ll be ready to start creating dashboards that automatically update themselves.

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form, or support other tools in performing, any appropriate action on these components . Eventually, it should be possible to give the system dependency list to the CDM and have it direct the appropriate parts of the BCVM to reload the repository, regenerate (in the right places) code and other software artifacts (makefiles, configuration files, and so on), recompile and relink the whole system, and even automatically rerun regression test-and all of this with one command . This may seem like dreamland, but a capability approaching this level has been created in the past by one of the authors . The CDM could also provide the following capabilities : Determine dependency consistency. That is, the CDM should be able to verify that there are no circular invocations and no business component participating twice at two different version levels . Consistency is determined by a set of rules that can change from project to project, since rules such as whether it is acceptable to find two different versions of the same DC in the system are project-dependent . However, managing this, where not disallowed for some technical reason, would mean introducing new concepts, such as "equivalence sets ." An equivalence set is a set of versions of a component that are considered equivalent for providing a given interface-a concept not addressed in this chapter . Starting from the dependency list, generate the appropriate artifacts (such as an IDE project file or makefile) used to build the entire business component system, and also build other "global" items such as the global database schema.

6. To decrease the opacity of the file in Video Track 2, use the Selection tool (the arrow icon above the hand icon) to click and drag the red rubberband (fade line) down. 7. To slowly fade a video clip, set the left side of the Opacity Rubberband so that it is at the top-left position and drag the far right side down, as shown in Figure 12-2. Figure 12-3 shows a few frames of the effect of a gradual fade. The fade in Figure 12-2 is gradual because the fade line gradually steps down. The top of the fade line indicates that the video clip is 100 percent opaque. When the fade line follows a long, slow, diagonal path from the top of the fade bar (100 percent opaque) to the bottom (100 percent transparent), the clip gradually fades out.

where OK,,t, is the unit tangential vector to the edge a, of K oriented compatibly with through the map XK. The tangential vectors t , are transformed by x~ according to the relation

produced by S. equi ssp. zooepidemicus 4881 (Akesson et al. 2007). It is composed of an N-terminal catalytic domain of putative endopeptidase activity and a C-terminal cell wall-binding domain (Lai et al. 2002). It was shown that zoocin A-susceptible streptococcal species include, aside from S. equi, S. pyogenes, Streptococcus mutans, and S. gordonii. It is worth noting that all ve S. pyogenes strains tested were extremely sensitive to zoocin A (MIC 31.5 ng/mL). Some other streptococcal species, especially S. oralis and S. rattus, are not susceptible to the enzyme s lytic activity (Akesson et al. 2007). Interestingly, zoocin A was found to be a penicillin-binding protein (PBP). In this regard, the enzyme was shown to bind penicillin covalently, to possess a weak -lactamase activity, and to contain motifs typical of other PBPs. Furthermore, incubation of zoocin A with penicillin decreased its enzymatic activity (Heath et al. 2004). Zoocin A was shown to be capable of killing S. mutans in a triple-species plaque model (this species is involved in the pathogenesis of dental caries; Simmonds et al. 1995).

Figure 1-11

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